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A sequence structure-based phylogeny of GPCR analysis suggests that n-arachidonylglycine is a potent ligand of GPR141 (Reference Kakarala and Jamil29)

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ActRIIB inhibition prevents muscle wasting and prolongs survival[56] via increased Akt activity, inactivation of FOXO3a and subsequent inhibition of Atrogin-1 and MuRF1[56]